Hi there!
I already posted this in the off-topic section and someone complained about it, being in the wrong thread. So i am going to post it here.
Is chess a dopmin driven feedback loop? Does dopamin rise when playing chess just like when taking drugs (cocaine), browsing social media and watching porn?
It would love to hear your opinion on this topic.
Thank you very much.
well yes
that exact thing is what life is about
unless you try to break the habit like the buddhism teaches
I think chess is very addictive I've been up playing since yester day at like 9 pm its 8 am now
Took the words outta my mouth!
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well, between the three things that you mentioned, the first and third ones have medical purposes. chess and social media don't.
dopamine shdopamine. Yes, sure only one neuro-transmitter, nothing else. With scrutiny into the wiring, one might realize that only looking at one part of the system does not make it the system.
It has even been called the pleasure loop (if not mistaken). For me it is involved in the effects of pleasure paths, which i don't know much about about. But I have read that random success was more effective than sustained success in committing an addiction deep in behaviour. And that dopamine level would be seen to increase in some regions of the brain in correlation something like that. correlation does not give causation, or if it does, it does not say in what direction or if direct.
If we were to separate motivation from pleasure (or other psychological causation factors) as distinct objects of study, we might have different view.
There are clear subjective link (in my subjectivity) between, a range of emotions, motivation, attention variants (sustained, selective, and what not that i am not aware of), pleasure, and degrees of productive concentration or obsessions (these point to the same thing in different dosage, i mean degrees). for me motivation and sustained attention are very much a daily storm.
The fact that dopamine is involved in motivation (documented and my subjectivity), does not make it part of the rest of the loop. Just that a lot of medication and pharmacology has been betting a lot on magic bullets, and prefer simple targets... or that was just the experimental constraints and knowledge. But it sure seems to have a momentum of its own, this mono-neurotransmitter behavioural explanation assumption.
